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شخصية إدمانية

من ويكيبيديا، الموسوعة الحرة
اذهب إلى التنقل اذهب إلى البحث
فهرس الإدمان والاعتمادية[1][2][3][4]

الشخصية الادمانية هي مجموعة السمات التي تجعل الشخص لديه استعداد للإدمان.[5][6][7] الأشخاص الذين لديهم اعتمادية على المواد يمكن تصنيفهم على أنهم لديهم اعتمادية على المواد بدرجة تؤثر سلبا على حياتهم. الإدمان مجرد سلوك نمطي برتبط بفعل معين يولد اللذة كالانترنت والعمل والمخدرات والتدخين وإلى آخره ويكمن الحل في أن يزاحم حياته بالأمور الأخرى الأقل امتاعا له.

مراجع[عدل]

  1. ^ Malenka RC، Nestler EJ، Hyman SE (2009). "Chapter 15: Reinforcement and Addictive Disorders". In Sydor A، Brown RY. Molecular Neuropharmacology: A Foundation for Clinical Neuroscience (الطبعة 2nd). New York: McGraw-Hill Medical. صفحات 364–375. ISBN 9780071481274. 
  2. ^ Nestler EJ (December 2013). "Cellular basis of memory for addiction". Dialogues Clin. Neurosci. 15 (4): 431–443. PMC 3898681Freely accessible. PMID 24459410. Despite the importance of numerous psychosocial factors, at its core, drug addiction involves a biological process: the ability of repeated exposure to a drug of abuse to induce changes in a vulnerable brain that drive the compulsive seeking and taking of drugs, and loss of control over drug use, that define a state of addiction. ... A large body of literature has demonstrated that such ΔFosB induction in D1-type [nucleus accumbens] neurons increases an animal's sensitivity to drug as well as natural rewards and promotes drug self-administration, presumably through a process of positive reinforcement ... Another ΔFosB target is cFos: as ΔFosB accumulates with repeated drug exposure it represses c-Fos and contributes to the molecular switch whereby ΔFosB is selectively induced in the chronic drug-treated state.41. ... Moreover, there is increasing evidence that, despite a range of genetic risks for addiction across the population, exposure to sufficiently high doses of a drug for long periods of time can transform someone who has relatively lower genetic loading into an addict. 
  3. ^ "Glossary of Terms". Mount Sinai School of Medicine. Department of Neuroscience. اطلع عليه بتاريخ 09 فبراير 2015. 
  4. ^ Volkow ND، Koob GF، McLellan AT (January 2016). "Neurobiologic Advances from the Brain Disease Model of Addiction". N. Engl. J. Med. 374 (4): 363–371. PMID 26816013. doi:10.1056/NEJMra1511480. Substance-use disorder: A diagnostic term in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) referring to recurrent use of alcohol or other drugs that causes clinically and functionally significant impairment, such as health problems, disability, and failure to meet major responsibilities at work, school, or home. Depending on the level of severity, this disorder is classified as mild, moderate, or severe.
    Addiction: A term used to indicate the most severe, chronic stage of substance-use disorder, in which there is a substantial loss of self-control, as indicated by compulsive drug taking despite the desire to stop taking the drug. In the DSM-5, the term addiction is synonymous with the classification of severe substance-use disorder.
     
  5. ^ Kourosh، Arianne S.؛ Harrington، Cynthia R.؛ Adinoff، Bryon (2010). "Tanning as a Behavioral Addiction". The American Journal of Drug and Alcohol Abuse. 36 (5): 284–90. PMID 20545604. doi:10.3109/00952990.2010.491883. 
  6. ^ Nealon، Dennis (October 3, 2012). "Adolescent psychiatrist talks about addictive personalities". University of Massachusetts Medical School. تمت أرشفته من الأصل في 09 فبراير 2013. اطلع عليه بتاريخ November 26, 2012. 
  7. ^ Dubreucq، Sarah؛ Koehl, Muriel؛ Abrous, Djoher N.؛ Marsicano, Giovanni؛ Chaouloff, Francis (July 2010). "CB1 receptor deficiency decreases wheel-running activity: Consequences on emotional behaviours and hippocampal neurogenesis". Experimental Neurology. 224 (1): 106–113. PMID 20138171. doi:10.1016/j.expneurol.2010.01.017. 


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