PRND
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هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
PRND (Prion like protein doppel) هوَ بروتين يُشَفر بواسطة جين PRND في الإنسان.[1]
الوظيفة[عدل]
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.الأهمية السريرية[عدل]
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.المراجع[عدل]
قراءة متعمقة[عدل]
- Moore RC، Lee IY، Silverman GL، وآخرون. (1999). "Ataxia in prion protein (PrP)-deficient mice is associated with upregulation of the novel PrP-like protein doppel.". J. Mol. Biol. 292 (4): 797–817. PMID 10525406. doi:10.1006/jmbi.1999.3108.
- Weissmann C، Aguzzi A (1999). "Perspectives: neurobiology. PrP's double causes trouble.". Science. 286 (5441): 914–5. PMID 10577243. doi:10.1126/science.286.5441.914.
- Peoc'h K، Guérin C، Brandel JP، وآخرون. (2000). "First report of polymorphisms in the prion-like protein gene (PRND): implications for human prion diseases.". Neurosci. Lett. 286 (2): 144–8. PMID 10825657. doi:10.1016/S0304-3940(00)01100-9.
- Lu K، Wang W، Xie Z، وآخرون. (2000). "Expression and structural characterization of the recombinant human doppel protein.". Biochemistry. 39 (44): 13575–83. PMID 11063595. doi:10.1021/bi001523m.
- Schröder B، Franz B، Hempfling P، وآخرون. (2001). "Polymorphisms within the prion-like protein gene (Prnd) and their implications in human prion diseases, Alzheimer's disease and other neurological disorders.". Hum. Genet. 109 (3): 319–25. PMID 11702213. doi:10.1007/s004390100591.
- Moore RC، Mastrangelo P، Bouzamondo E، وآخرون. (2002). "Doppel-induced cerebellar degeneration in transgenic mice.". Proc. Natl. Acad. Sci. U.S.A. 98 (26): 15288–93. PMC 65022
. PMID 11734625. doi:10.1073/pnas.251550798. - Deloukas P، Matthews LH، Ashurst J، وآخرون. (2002). "The DNA sequence and comparative analysis of human chromosome 20.". Nature. 414 (6866): 865–71. PMID 11780052. doi:10.1038/414865a.
- Peoc'h K، Serres C، Frobert Y، وآخرون. (2003). "The human "prion-like" protein Doppel is expressed in both Sertoli cells and spermatozoa.". J. Biol. Chem. 277 (45): 43071–8. PMID 12200435. doi:10.1074/jbc.M206357200.
- Infante J، Llorca J، Rodero L، وآخرون. (2003). "Polymorphism at codon 174 of the prion-like protein gene is not associated with sporadic Alzheimer's disease.". Neurosci. Lett. 332 (3): 213–5. PMID 12399017. doi:10.1016/S0304-3940(02)00941-2.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون. (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences.". Proc. Natl. Acad. Sci. U.S.A. 99 (26): 16899–903. PMC 139241. PMID 12477932. doi:10.1073/pnas.242603899.
- Makrinou E، Collinge J، Antoniou M (2003). "Genomic characterization of the human prion protein (PrP) gene locus.". Mamm. Genome. 13 (12): 696–703. PMID 12514748. doi:10.1007/s00335-002-3043-0.
- Peoc'h K، Volland H، De Gassart A، وآخرون. (2003). "Prion-like protein Doppel expression is not modified in scrapie-infected cells and in the brains of patients with Creutzfeldt–Jakob disease.". FEBS Lett. 536 (1–3): 61–5. PMID 12586339. doi:10.1016/S0014-5793(03)00012-7.
- Lührs T، Riek R، Güntert P، Wüthrich K (2003). "NMR structure of the human doppel protein". J. Mol. Biol. 326 (5): 1549–57. PMID 12595265. doi:10.1016/S0022-2836(02)01471-7.
- Clark HF، Gurney AL، Abaya E، وآخرون. (2003). "The secreted protein discovery initiative (SPDI), a large-scale effort to identify novel human secreted and transmembrane proteins: a bioinformatics assessment". Genome Res. 13 (10): 2265–70. PMC 403697. PMID 12975309. doi:10.1101/gr.1293003.
- Hundt C، Weiss S (2004). "The prion-like protein Doppel fails to interact with itself, the prion protein and the 37 kDa/67 kDa laminin receptor in the yeast two-hybrid system". Biochim. Biophys. Acta. 1689 (1): 1–5. PMID 15158907. doi:10.1016/j.bbadis.2004.02.003.
- Massimino ML، Ballarin C، Bertoli A، وآخرون. (2005). "Human Doppel and prion protein share common membrane microdomains and internalization pathways". Int. J. Biochem. Cell Biol. 36 (10): 2016–31. PMID 15203115. doi:10.1016/j.biocel.2004.03.002.
- Yin SM، Sy MS، Yang HY، Tien P (2004). "Interaction of Doppel with the full-length laminin receptor precursor protein". Arch. Biochem. Biophys. 428 (2): 165–9. PMID 15246873. doi:10.1016/j.abb.2004.06.003.
- Gerhard DS، Wagner L، Feingold EA، وآخرون. (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. 14 (10B): 2121–7. PMC 528928. PMID 15489334. doi:10.1101/gr.2596504.
- Serres C، Peoc'h K، Courtot AM، وآخرون. (2006). "Spatio-developmental distribution of the prion-like protein doppel in Mammalian testis: a comparative analysis focusing on its presence in the acrosome of spermatids". Biol. Reprod. 74 (5): 816–23. PMID 16421231. doi:10.1095/biolreprod.105.047829.
- Azzalin A، Del Vecchio I، Ferretti L، Comincini S (2007). "The prion-like protein Doppel (Dpl) interacts with the human receptor for activated C-kinase 1 (RACK1) protein". Anticancer Res. 26 (6B): 4539–47. PMID 17201176.
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