FOSL2
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هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
FOSL2 (FOS like 2, AP-1 transcription factor subunit) هوَ بروتين يُشَفر بواسطة جين FOSL2 في الإنسان.[1]
الوظيفة[عدل]

الأهمية السريرية[عدل]

المراجع[عدل]
- ^ "Entrez Gene: FOSL2 FOS-like antigen 2". مؤرشف من الأصل في 05 ديسمبر 2010.
قراءة متعمقة[عدل]
- Matsui M، Tokuhara M، Konuma Y، Nomura N، Ishizaki R (March 1990). "Isolation of human fos-related genes and their expression during monocyte-macrophage differentiation". Oncogene. 5 (3): 249–55. PMID 2107490.
- Molven A، Houge G، Berger R (November 1996). "Chromosomal assignment of the human gene encoding the Fos-related antigen-2 (FRA2) to chromosome 2p22-p23". Genomics. 38 (1): 72–5. PMID 8954781. doi:10.1006/geno.1996.0593.
- Outinen PA، Sood SK، Pfeifer SI، Pamidi S، Podor TJ، Li J، Weitz JI، Austin RC (August 1999). "Homocysteine-induced endoplasmic reticulum stress and growth arrest leads to specific changes in gene expression in human vascular endothelial cells". Blood. 94 (3): 959–67. PMID 10419887.
- Miyamoto NG، Medberry PS، Hesselgesser J، Boehlk S، Nelson PJ، Krensky AM، Perez HD (June 2000). "Interleukin-1beta induction of the chemokine RANTES promoter in the human astrocytoma line CH235 requires both constitutive and inducible transcription factors". Journal of Neuroimmunology. 105 (1): 78–90. PMID 10713367. doi:10.1016/S0165-5728(00)00195-8.
- Ng DC، Shafaee S، Lee D، Bikle DD (August 2000). "Requirement of an AP-1 site in the calcium response region of the involucrin promoter". The Journal of Biological Chemistry. 275 (31): 24080–8. PMID 10816578. doi:10.1074/jbc.M002508200.
- Bamberger AM، Milde-Langosch K، Rössing E، Goemann C، Löning T (September 2001). "Expression pattern of the AP-1 family in endometrial cancer: correlations with cell cycle regulators". Journal of Cancer Research and Clinical Oncology. 127 (9): 545–50. PMID 11570575. doi:10.1007/s004320100255.
- Udalova IA، Kwiatkowski D (November 2001). "Interaction of AP-1 with a cluster of NF-kappa B binding elements in the human TNF promoter region". Biochemical and Biophysical Research Communications. 289 (1): 25–33. PMID 11708771. doi:10.1006/bbrc.2001.5929.
- Benkoussa M، Brand C، Delmotte MH، Formstecher P، Lefebvre P (July 2002). "Retinoic acid receptors inhibit AP1 activation by regulating extracellular signal-regulated kinase and CBP recruitment to an AP1-responsive promoter". Molecular and Cellular Biology. 22 (13): 4522–34. PMC 133906
. PMID 12052862. doi:10.1128/MCB.22.13.4522-4534.2002.
- Santalucía T، Christmann M، Yacoub MH، Brand NJ (September 2003). "Hypertrophic agonists induce the binding of c-Fos to an AP-1 site in cardiac myocytes: implications for the expression of GLUT1". Cardiovascular Research. 59 (3): 639–48. PMID 14499865. doi:10.1016/S0008-6363(03)00472-3.
- Beausoleil SA، Jedrychowski M، Schwartz D، Elias JE، Villén J، Li J، Cohn MA، Cantley LC، Gygi SP (August 2004). "Large-scale characterization of HeLa cell nuclear phosphoproteins". Proceedings of the National Academy of Sciences of the United States of America. 101 (33): 12130–5. PMC 514446
. PMID 15302935. doi:10.1073/pnas.0404720101.
- Olsen JV، Blagoev B، Gnad F، Macek B، Kumar C، Mortensen P، Mann M (November 2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. 127 (3): 635–48. PMID 17081983. doi:10.1016/j.cell.2006.09.026.
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