PSME4
اذهب إلى التنقل
اذهب إلى البحث
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
هذا القسم فارغ أو غير مكتمل، ساهم بتحريره.
PSME4 (Proteasome activator subunit 4) هوَ بروتين يُشَفر بواسطة جين PSME4 في الإنسان.[1][2][3]
الوظيفة[عدل]

الأهمية السريرية[عدل]

المراجع[عدل]
- ^ Ustrell V، Hoffman L، Pratt G، Rechsteiner M (Jul 2002). "PA200, a nuclear proteasome activator involved in DNA repair". The EMBO Journal. 21 (13): 3516–25. PMC 126083
. PMID 12093752. doi:10.1093/emboj/cdf333.
- ^ Nomura N، Nagase T، Miyajima N، Sazuka T، Tanaka A، Sato S، Seki N، Kawarabayasi Y، Ishikawa K، Tabata S (Dec 1995). "Prediction of the coding sequences of unidentified human genes. II. The coding sequences of 40 new genes (KIAA0041-KIAA0080) deduced by analysis of cDNA clones from human cell line KG-1". DNA Research. 1 (5): 223–9. PMID 7584044. doi:10.1093/dnares/1.5.223.
- ^ "Entrez Gene: PSME4 proteasome (prosome, macropain) activator subunit 4". مؤرشف من الأصل في 05 ديسمبر 2010.
قراءة متعمقة[عدل]
- Goff SP (Aug 2003). "Death by deamination: a novel host restriction system for HIV-1". Cell. 114 (3): 281–3. PMID 12914693. doi:10.1016/S0092-8674(03)00602-0.
- Seeger M، Ferrell K، Frank R، Dubiel W (Mar 1997). "HIV-1 tat inhibits the 20 S proteasome and its 11 S regulator-mediated activation". The Journal of Biological Chemistry. 272 (13): 8145–8. PMID 9079628. doi:10.1074/jbc.272.13.8145.
- Madani N، Kabat D (Dec 1998). "An endogenous inhibitor of human immunodeficiency virus in human lymphocytes is overcome by the viral Vif protein". Journal of Virology. 72 (12): 10251–5. PMC 110608
. PMID 9811770.
- Simon JH، Gaddis NC، Fouchier RA، Malim MH (Dec 1998). "Evidence for a newly discovered cellular anti-HIV-1 phenotype". Nature Medicine. 4 (12): 1397–400. PMID 9846577. doi:10.1038/3987.
- Mulder LC، Muesing MA (Sep 2000). "Degradation of HIV-1 integrase by the N-end rule pathway". The Journal of Biological Chemistry. 275 (38): 29749–53. PMID 10893419. doi:10.1074/jbc.M004670200.
- Sheehy AM، Gaddis NC، Choi JD، Malim MH (Aug 2002). "Isolation of a human gene that inhibits HIV-1 infection and is suppressed by the viral Vif protein". Nature. 418 (6898): 646–50. Bibcode:2002Natur.418..646S. PMID 12167863. doi:10.1038/nature00939.
- Huang X، Seifert U، Salzmann U، Henklein P، Preissner R، Henke W، Sijts AJ، Kloetzel PM، Dubiel W (Nov 2002). "The RTP site shared by the HIV-1 Tat protein and the 11S regulator subunit alpha is crucial for their effects on proteasome function including antigen processing". Journal of Molecular Biology. 323 (4): 771–82. PMID 12419264. doi:10.1016/S0022-2836(02)00998-1.
- Gaddis NC، Chertova E، Sheehy AM، Henderson LE، Malim MH (May 2003). "Comprehensive investigation of the molecular defect in vif-deficient human immunodeficiency virus type 1 virions". Journal of Virology. 77 (10): 5810–20. PMC 154025
. PMID 12719574. doi:10.1128/JVI.77.10.5810-5820.2003.
- Lecossier D، Bouchonnet F، Clavel F، Hance AJ (May 2003). "Hypermutation of HIV-1 DNA in the absence of the Vif protein". Science. 300 (5622): 1112. PMID 12750511. doi:10.1126/science.1083338.
- Zhang H، Yang B، Pomerantz RJ، Zhang C، Arunachalam SC، Gao L (Jul 2003). "The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA". Nature. 424 (6944): 94–8. Bibcode:2003Natur.424...94Z. PMC 1350966
. PMID 12808465. doi:10.1038/nature01707.
- Mangeat B، Turelli P، Caron G، Friedli M، Perrin L، Trono D (Jul 2003). "Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcripts". Nature. 424 (6944): 99–103. Bibcode:2003Natur.424...99M. PMID 12808466. doi:10.1038/nature01709.
- Harris RS، Bishop KN، Sheehy AM، Craig HM، Petersen-Mahrt SK، Watt IN، Neuberger MS، Malim MH (Jun 2003). "DNA deamination mediates innate immunity to retroviral infection". Cell. 113 (6): 803–9. PMID 12809610. doi:10.1016/S0092-8674(03)00423-9.
- Harris RS، Sheehy AM، Craig HM، Malim MH، Neuberger MS (Jul 2003). "DNA deamination: not just a trigger for antibody diversification but also a mechanism for defense against retroviruses". Nature Immunology. 4 (7): 641–3. PMID 12830140. doi:10.1038/ni0703-641.
- Gu Y، Sundquist WI (Jul 2003). "Good to CU". Nature. 424 (6944): 21–2. Bibcode:2003Natur.424...21G. PMID 12840737. doi:10.1038/424021a.
- Mariani R، Chen D، Schröfelbauer B، Navarro F، König R، Bollman B، Münk C، Nymark-McMahon H، Landau NR (Jul 2003). "Species-specific exclusion of APOBEC3G from HIV-1 virions by Vif". Cell. 114 (1): 21–31. PMID 12859895. doi:10.1016/S0092-8674(03)00515-4.
- KewalRamani VN، Coffin JM (Aug 2003). "Virology. Weapons of mutational destruction". Science. 301 (5635): 923–5. PMID 12920286. doi:10.1126/science.1088965.
|
|
